In this issue of Blood, Casu et al report on results from their studies on potential therapeutic options to restrict iron as a treatment of 2 differing blood disorders: the anemia and iron overload associated with b-thalassemia, and the erythrocytosis associated with polycythemia vera (PV). They examine the use of derivatives of hepcidin, a 25-amino acid peptide secreted by the liver, which is widely accepted as the "iron stores" regulator.(1)
Authors | Subramaniam, V. Nathan |
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Journal | BLOOD |
Pages | 153-154 |
Volume | 128 |
Date | 1/07/2016 |
Grant ID | |
Funding Body | |
URL | http://www.ncbi.nlm.nih.gov/pubmed/?term=10.1182/blood-2016-05-716043 |
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